AAO Academy Online

This content package represents several years of education, accessible in 2018 and early 2019.

This session is a requirement for students participating in the Student Fellowship program at Academy 2019 Orlando and 3rd World Congress of Optometry, October 23-27. You must login with your Academy credentials to view the orientation video and complete the required quiz at the end. This orientation video must be completed by October 28, 2019 at 11:59 PM ET.

This presentation provides necessary understanding of the signs and symptoms that accompany various types of neurogenic diplopia. An additional goal is to understand possible systemic implications and appropriate medical testing for patients presenting with diplopia. This course details in a case based format the diagnosis of patients presenting with diplopia. A diagnostic algorithm is presented to allow the audience member to follow the clinical findings to an appropriate differential diagnosis with emphasis on pertinent neuroanatomy and potential causative factors. Current therapeutic management and appropriate diagnostic testing is heavily emphasized and key points are reinforced with easy-to-remember 'Odes'.

You will gain a comprehensive review of AMD and the differentiation of “dry” and “wet”. You will learn the results of the latest clinical trials and an update on new technology that have changed the perspective on how patients with macular degeneration are managed. There has been an explosion of new options that you will be able to implement immediately as well as update you on future treatment modalities.

This course focuses on the history, associated symptoms, ophthalmic examination, differential diagnosis, and work-up for the patient with transient monocular and binocular visual loss. Ocular and systemic etiologies are reviewed

Giant cell arteritis (GCA) regularly presents to eye care providers. While anterior ischemic optic neuropathy is the most common ocular manifestation of GCA, amaurosis fugax, retinal artery occlusions, cranial nerve palsies, and posterior ischemic optic neuropathy are also sequelae. Early diagnosis and urgent treatment of GCA is required to prevent possible bilateral blindness. Standards of care for the more common etiologies of the ocular manifestations of GCA may not involve the same urgency of intervention. Therefore, optometrists are placed in the difficult situation of determining the likelihood of GCA being the underlying cause of these relatively common ocular diagnoses.